Associated Papers
Wilms tumor primary cultures capture phenotypic heterogeneity and facilitate preclinical screening.
Götz L et al.
Translational Oncology, 2025
TRIM28 inactivation in epithelial nephroblastoma is frequent and often associated with predisposing TRIM28 germline variants.
Wegert J et al.
The Journal of Pathology, 2024
TRIM28 variants and Wilms' tumour predisposition.
Hol JA et al.
The Journal of Pathology, 2021
TRIM28 congenital predisposition to Wilms' tumor: novel mutations and presentation in a sibling pair.
Moore C et al.
Cold Spring Harbor Molecular Case Studies, 2020
Identification of new Wilms tumour predisposition genes: an exome sequencing study.
Mahamdallie S et al.
The Lancet. Child & Adolescent Health, 2019
A unique subset of low-risk Wilms tumors is characterized by loss of function of TRIM28 (KAP1), a gene critical in early renal development: A Children's Oncology Group study.
Armstrong AE et al.
Plos One, 2018
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