Associated Papers
Elevated ubiquitin phosphorylation by PINK1 contributes to proteasomal impairment and promotes neurodegeneration.
Chen C et al.
Elife, 2025
AMPK Promotes SPOP-Mediated NANOG Degradation to Regulate Prostate Cancer Cell Stemness.
Wang X et al.
Developmental Cell, 2019
(Patho-)physiological relevance of PINK1-dependent ubiquitin phosphorylation.
Fiesel FC et al.
Embo Reports, 2015
Phosphorylation of Parkin at Serine65 is essential for activation: elaboration of a Miro1 substrate-based assay of Parkin E3 ligase activity.
Kazlauskaite A et al.
Open Biology, 2014
24647965 (PubMed )
Supplement: rsob130213supp1.pdf
Inactivation of VHL by tumorigenic mutations that disrupt dynamic coupling of the pVHL.hypoxia-inducible transcription factor-1alpha complex.
Miller F et al.
The Journal of Biological Chemistry, 2005
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