Associated Papers
Progress on the mechanism of Polycystin-1 in bone remodeling.
Guo J et al.
Frontiers in Cell and Developmental Biology, 2025
The L27 domain of MPP7 enhances TAZ-YY1 cooperation to renew muscle stem cells.
Shao A et al.
Embo Reports, 2024
LATS2 condensates organize signalosomes for Hippo pathway signal transduction.
Qin M et al.
Nature Chemical Biology, 2024
Epithelioid hemangioendothelioma (EHE) with WWTR1::TFE3 gene fusion, a novel fusion variant.
Li S et al.
Genes, Chromosomes & Cancer, 2024
The L27 Domain of MPP7 enhances TAZ-YY1 Cooperation to Renew Muscle Stem Cells.
Shao A et al.
Biorxiv : the Preprint Server for Biology, 2023
TGF-β/SMAD canonical pathway induces the expression of transcriptional cofactor TAZ in liver cancer cells.
Ríos-López DG et al.
Heliyon, 2023
Distinctive Roles of YAP and TAZ in Human Endothelial Progenitor Cells Growth and Functions.
Klaihmon P et al.
Biomedicines, 2022
TAZ-CAMTA1 and YAP-TFE3 alter the TAZ/YAP transcriptome by recruiting the ATAC histone acetyltransferase complex.
Merritt N et al.
Elife, 2021
SKI activates the Hippo pathway via LIMD1 to inhibit cardiac fibroblast activation.
Landry NM et al.
Basic Research in Cardiology, 2021
WWTR1(TAZ)-CAMTA1 reprograms endothelial cells to drive epithelioid hemangioendothelioma.
Driskill JH et al.
Genes & Development, 2021
miR-582-5p Is a Tumor Suppressor microRNA Targeting the Hippo-YAP/TAZ Signaling Pathway in Non-Small Cell Lung Cancer.
Zhu B et al.
Cancers, 2021
Polycystin-1 regulates bone development through an interaction with the transcriptional coactivator TAZ.
Merrick D et al.
Human Molecular Genetics, 2019
Actin like-6A promotes glioma progression through stabilization of transcriptional regulators YAP/TAZ.
Ji J et al.
Cell Death & Disease, 2018
KIBRA (WWC1) Is a Metastasis Suppressor Gene Affected by Chromosome 5q Loss in Triple-Negative Breast Cancer.
Knight JF et al.
Cell Reports, 2018
Transforming Growth Factor-β Promotes Liver Tumorigenesis in Mice via Up-regulation of Snail.
Moon H et al.
Gastroenterology, 2017
YAP/TAZ-mediated activation of serine metabolism and methylation regulation is critical for LKB1-deficient breast cancer progression.
Wu Q et al.
Bioscience Reports, 2017
STK38L kinase ablation promotes loss of cell viability in a subset of KRAS-dependent pancreatic cancer cell lines.
Grant TJ et al.
Oncotarget, 2017
Reciprocal stabilization of ABL and TAZ regulates osteoblastogenesis through transcription factor RUNX2.
Matsumoto Y et al.
The Journal of Clinical Investigation, 2016
Wnt Signaling Promotes Breast Cancer by Blocking ITCH-Mediated Degradation of YAP/TAZ Transcriptional Coactivator WBP2.
Lim SK et al.
Cancer Research, 2016
CDK1 phosphorylation of TAZ in mitosis inhibits its oncogenic activity.
Zhang L et al.
Oncotarget, 2015
Ski regulates Hippo and TAZ signaling to suppress breast cancer progression.
Rashidian J et al.
Science Signaling, 2015
The Hippo transducer TAZ interacts with the SWI/SNF complex to regulate breast epithelial lineage commitment.
Skibinski A et al.
Cell Reports, 2014
Differential topical susceptibility to TGFβ in intact and injured regions of the epithelium: key role in myofibroblast transition.
Speight P et al.
Molecular Biology of the Cell, 2013
YAP and TAZ, Hippo signaling targets, act as a rheostat for nuclear SHP2 function.
Tsutsumi R et al.
Developmental Cell, 2013
Cellular localization and characterization of cytosolic binding partners for Gla domain-containing proteins PRRG4 and PRRG2.
Yazicioglu MN et al.
The Journal of Biological Chemistry, 2013
Angiomotin family proteins are novel activators of the LATS2 kinase tumor suppressor.
Paramasivam M et al.
Molecular Biology of the Cell, 2011
WW domain-mediated interaction with Wbp2 is important for the oncogenic property of TAZ.
Chan SW et al.
Oncogene, 2011
A novel mechanism of cell growth regulation by Cell Cycle and Apoptosis Regulatory Protein (CARP)-1.
Jiang Y et al.
Journal of Molecular Signaling, 2010
TEADs mediate nuclear retention of TAZ to promote oncogenic transformation.
Chan SW et al.
The Journal of Biological Chemistry, 2009
TAZ: a novel transcriptional co-activator regulated by interactions with 14-3-3 and PDZ domain proteins.
Kanai F et al.
The Embo Journal, 2000