Associated Papers
ZP1-Y262C mutation causes abnormal zona pellucida formation and female infertility in humans.
Cao G et al.
Frontiers in Genetics, 2024
Novel variants in ZP1, ZP2 and ZP3 associated with empty follicle syndrome and abnormal zona pellucida.
Sun L et al.
Reproductive Biomedicine Online, 2023
Gene mutations impede oocyte maturation, fertilization, and early embryonic development.
Fei CF et al.
Bioessays : News and Reviews in Molecular, Cellular and Developmental Biology, 2022
Novel biallelic loss-of-function variants in ZP1 identified in an infertile female with empty follicle syndrome.
Liu M et al.
Journal of Assisted Reproduction and Genetics, 2020
Homozygous Splice Site Mutation in ZP1 Causes Familial Oocyte Maturation Defect.
Okutman Ö et al.
Genes, 2020
ZP1 mutations are associated with empty follicle syndrome: evidence for the existence of an intact oocyte and a zona pellucida in follicles up to the early antral stage. A case report.
Dai C et al.
Human Reproduction (oxford, England), 2019
[The human zona pellucida: New genetic findings].
Trocka D et al.
Medecine Sciences : M/s, 0
Genetic variations in zona pellucida glycoproteins: Implications for fertility and ART outcomes.
Rajput N et al.
Mutation Research. Reviews in Mutation Research, 0
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