Associated Papers
A Catalytically Inactive Protein Kinase C alpha Mutation Drives Chordoid Glioma by Pathway Rewiring.
Bellamy C et al.
Biorxiv : the Preprint Server for Biology, 2025
The complexities of PKCα signaling in cancer.
Black AR et al.
Advances in Biological Regulation, 2021
A recurrent point mutation in PRKCA is a hallmark of chordoid gliomas.
Rosenberg S et al.
Nature Communications, 2018
Reversing the Paradigm: Protein Kinase C as a Tumor Suppressor.
Newton AC et al.
Trends in Pharmacological Sciences, 2017
Cancer-associated protein kinase C mutations reveal kinase's role as tumor suppressor.
Antal CE et al.
Cell, 2015
Protein kinase signaling networks in cancer.
Brognard J et al.
Current Opinion in Genetics & Development, 2011
The PKCalpha-D294G mutant found in pituitary and thyroid tumors fails to transduce extracellular signals.
Zhu Y et al.
Cancer Research, 2005
Isozyme-specific abnormalities of PKC in thyroid cancer: evidence for post-transcriptional changes in PKC epsilon.
Knauf JA et al.
The Journal of Clinical Endocrinology and Metabolism, 2002
Selective loss of substrate recognition induced by the tumour-associated D294G point mutation in protein kinase Calpha.
Prévostel C et al.
The Biochemical Journal, 1998
Protein kinase C alpha and tumorigenesis of the endocrine gland.
Prévostel C et al.
Hormone Research, 1997
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