Associated Papers
The Effect of Treatment-Associated Mutations on HIV Replication and Transmission Cycles.
Johnson MM et al.
Viruses, 2022
Spontaneous Mutations in HIV-1 Gag, Protease, RT p66 in the First Replication Cycle and How They Appear: Insights from an In Vitro Assay on Mutation Rates and Types.
Yeo JY et al.
International Journal of Molecular Sciences, 2020
33396460 (PubMed )
Supplement: ijms-22-00370-s001.pdf
Frequent emergence of N348I in HIV-1 subtype C reverse transcriptase with failure of initial therapy reduces susceptibility to reverse-transcriptase inhibitors.
Brehm JH et al.
Clinical Infectious Diseases : an Official Publication of the Infectious Diseases Society of America, 2012
Clinical, virological and biochemical evidence supporting the association of HIV-1 reverse transcriptase polymorphism R284K and thymidine analogue resistance mutations M41L, L210W and T215Y in patients failing tenofovir/emtricitabine therapy.
Betancor G et al.
Retrovirology, 2012
Characterization and structural analysis of novel mutations in human immunodeficiency virus type 1 reverse transcriptase involved in the regulation of resistance to nonnucleoside inhibitors.
Ceccherini-Silberstein F et al.
Journal of Virology, 2007
Genotypic correlates of phenotypic resistance to efavirenz in virus isolates from patients failing nonnucleoside reverse transcriptase inhibitor therapy.
Bacheler L et al.
Journal of Virology, 2001
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