Associated Papers
The Clinical and Genetic Features in Chinese Children With Steroid-Resistant or Early-Onset Nephrotic Syndrome: A Multicenter Cohort Study.
Zhu X et al.
Frontiers in Medicine, 2022
35755072 (PubMed )
Supplement: Data_Sheet_1.PDF
Mutations in WT1 in boys with sporadic isolated steroid-resistant nephrotic syndrome.
Yang Y et al.
Genetics and Molecular Research : Gmr, 2016
Bilateral Wilms tumor and early presentation in pediatric patients is associated with the truncation of the Wilms tumor 1 protein.
Hu M et al.
The Journal of Pediatrics, 2013
[Clinical and pathological features of Denys-Drash syndrome: report of 3 cases].
Wang HY et al.
Zhonghua Er Ke Za Zhi = Chinese Journal of Pediatrics, 2012
Discordant phenotypes in monozygotic twins with identical de novo WT1 mutation.
Yu Z et al.
Clinical Kidney Journal, 2012
Wilms' tumor 1 single-nucleotide polymorphism rs16754 does not predict clinical outcome in adult acute myeloid leukemia.
Renneville A et al.
Leukemia, 2011
Clinical relevance of Wilms tumor 1 gene mutations in childhood acute myeloid leukemia.
Hollink IH et al.
Blood, 2009
Mutations in the Wilms' tumor 1 gene cause isolated steroid resistant nephrotic syndrome and occur in exons 8 and 9.
Mucha B et al.
Pediatric Research, 2006
Clinical features and an atypical WT1 mutant site in a child with incomplete Denys-Drash syndrome.
Wang HY et al.
Asian Journal of Andrology, 0
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